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J Comp Neurol 403(2):261280 Kieffer TJ, McIntosh CH, Pederson RA (1995) Degradation of glucose-dependent insulinotropic polypeptide and truncated glucagon-like peptide 1 in vitro and in vivo by dipeptidyl peptidase IV
The problem is that this fat-burning process generates a large amount of reactive oxygen species, which can damage cells
Its smaller molecular structure allows efficient diffusion in tissue models, facilitating localized cellular interactions
This cycle is amplified by copper's ability to catalyze the oxidation of catecholamines, generating additional toxic quinones and ROS that contribute to cardiotoxicity [113]
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