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This redox imbalance promotes mitochondrial DNA damage, lipid peroxidation, and activation of apoptotic pathways, ultimately contributing to myofibrillar disorganisation and interstitial fibrosis (73)

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Gene expression changes were calculated using the comparative CT (CT) method, normalized to GAPDH expression, and compared to cell lysates from soft substrates coated with COL IV

Resolution of NASH and hepatic fibrosis by the GLP-1R/GcgR dual-agonist Cotadutide via modulating mitochondrial function and lipogenesis