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When DNA is broken, a series of molecules in the DNA damage response pathway are activated, which first includes the ataxia-telangiectasia-mutated (ATM) and ataxia telangiectasia and rad3-related (ATR) kinases and leads to the p53 tumor suppressor protein through CHK1 and CHK2 (6, 7)

ID8 Trp53 / ovarian cancer cells (derived from C57BL/6 mouse) have been described before 57

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The GH selectivity profile was established in animal models

The human organism can synthesize it itself from the three amino acids mentioned, but optimal availability is subject to several conditions