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An increase in overall mitochondrial ROS generation will have a significant impact on cellular redox balance ( 3.2 ROS-mediated inflammatory signaling pathways NF-B is a key regulator of inflammatory and immune responses and increases the expression of pro-inflammatory cytokines such as TNF- and IL-1 by up-regulating gene expression through enhanced transcription activating mRNA stabilization ( 2 O 2 -mediated aryl hydrocarbon receptor nuclear translocation and A20 inhibition, providing a positive feedback loop for increased ROS and pro-inflammatory gene expression (Figure 2)

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