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The upregulation of TXN and TXNRD in cancer cells enhances their proliferative and resistance to apoptosis, underscoring the TXNTXNRD system as a promising target for therapeutic intervention [52, 53]

These combined effects may make DSIP a potential therapeutic tool for stress management

This gap underscores the need for comprehensive comparative studies to gain a deeper understanding of how peptide size and complexity impact therapeutic outcomes in wound healing

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These include natural ways of preventing mast cells from degranulating, either by avoiding triggers or substances that block various aspects of mast cell degranulation, as well as the research on medications and testing options