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Simon, M

Although our primary hypothesis concerns glutathione and glutamate, the underlying hypothesis is that in the residual state there is diminished neural metabolic integrity arising from a toxic process in the acute phase of illness [4]

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It may also be possible that the exposure conditions on the plaster of Paris substrate used by Sillapawattana and Schffer (2017) were different from those in the LUFA 2.2 soil, further adding to the differences in the response of the springtails to imidacloprid