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however, disruption of mitochondrial iron homeostasis enlarges the labile iron pool (LIP), intensifying ROS production through iron-catalyzed reactions

In the blood, Fe 3+ binds to transferrin (Tf) to form a complex which can be delivered into the cells by binding to transferrin receptor-1 (TFR1) in the cell membrane which is then transported to the endosome ( 3+ is converted to Fe 2+ by oxidation-reduction and is then released into a labile iron pool in the cytoplasm

However, off-target effects still cause the risk of normal cell damage and serious side effects

In conjunction with other cytokines such as TNF- and IL-6, this forms a critical axis impacting ferroptosis in inflammatory and tumor immune environments