Jones DP, Coates RJ, Flagg EW, et al
These findings demonstrate that VPA exposure induces significant microglial activation in the hippocampal CA1 region, which is effectively attenuated by hCiPSC-NSCs treatment, indicating a substantial reduction in neuroinflammatory responses
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Oxidative damage has been linked to high levels of eryptosis through an increase in the intracellular free Ca 2+ concentration, as evidenced by in vitro studies [24, 134, 300, 301] in patients intoxicated with lead [302], and in diabetic patients with chronic renal damage [303]
