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Table 2 Comparison of Spinal MMNST and SSP-MMNST

This initiates the ULK1 complex, promoting autophagosome formation and selective removal of damaged mitochondria through mitophagy (PINK1/Parkin pathway)

The clinical evidence base supports its use as a tool for maintaining antioxidant status, supporting liver and immune function, and reducing oxidative burden, particularly in individuals whose endogenous production is compromised by age, chronic stress, illness, or inadequate nutrient precursor availability