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These are practical strategies, not crutches

It is, therefore, reasonable to consider that the postprandial rise in GLP-1 might affect feeding behavior after RYGB, and to a lesser extent SG, where the increase in GLP-1 is less marked (72) have shown that inhibition of GLP-1 actions in the postprandial period has limited effects on glucose concentrations in people after RYGB

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In the rodent high fat fed male mice model, administration of semaglutide at the dose of 2800 g/kg/day resulted in a significant weight loss attributed to a decreased food intake [70]

Both affect the same system but through mechanisms producing wildly different results