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10.1172/JCI29877 16 BachettiT.CominiL.PasiniE.CargnoniA.CurelloS.FerrariR

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This dose-dependent slowing of gastric emptying represents a major mechanism contributing to PPG lowering by exogenous GLP-1 and, in healthy humans, outweighs its insulinotropic effects such that postprandial insulin secretory responses are suppressed rather than stimulated (15, 16)