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Anti-apoptotic effects of EPO (targeting primarily CFU-e and proerythroblasts) are mediated via pro-survival PI3K/Akt signaling activation [104], JAK2/STAT5-dependent upregulation of anti-apoptotic Bcl-XL [105, 106], JAK2/STAT5-dependent downregulation of pro-apoptotic Fas, FasL and BH3-only protein Bim [103], activation of the CK1-dependent caspase-10-P13tBid axis [107], and ERK-dependent degradation of Bim [108], summarized in Fig

The effectivity of BBB-specific cellular internalization and permeability can be increased by the combination of different targeting ligands on the surface of nanocarriers

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NEAT1-FTO negative regulation:FTO demethylates the m 6 A site of NEAT1, destroys its binding to HNRNPK, leads to NEAT1 degradation, releases the isolation of SFPQ, and inhibits RRM2 transcription (Knutsen et al., 2022)