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We noticed that the dose of testosterone as small as 0.08 mg/kg/day, which produces a small increase of the circulating hormone, could stimulate IGF-1 to reach pubertal concentrations and increase growth velocity to values comparable to the peak growth velocity (Table 1)

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In this Review, we highlight current understanding of the direct and indirect antiinflammatory effects and mechanisms of GLP-1 medicines in both preclinical and clinical studies, covering emerging concepts, clinical relevance, and areas of uncertainty that require further investigation

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