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Curcumin can also compete with LPS for TLR4 receptor activation, thereby inhibiting the TLR4/myeloid differentiation 88 (MyD88)/NF-B signaling pathways (150, Oral supplementation with curcumin reduces OS generated by hyperthyroidism in rats, as shown by decreased LPx and protein carbonyl (PC) levels and increased SOD and CAT activities in tissues ( In vitro studies show that curcumin can boost cellular glutathione levels by stimulating the transcription of two Gcl genes ( Gclc and Gclm ) encoding glutamate cysteine ligase, which is the rate-limiting enzyme in glutathione synthesis ( Supplementary Table 1 )

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