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In summary, the harsh metabolic landscape of the TME, characterized by nutrient competition, accumulation of immunosuppressive metabolites, and dysregulated lipid and amino acid metabolism, severely impairs the function and fate of infiltrating immune cells, ultimately promoting tumor immune evasion and progression

GLP-1 receptor agonists are designed with varying half-lives, which determine how long the drug remains therapeutically active in the body

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They bind to GLP-1 receptors located throughout the body, including the pancreas, brain, gastrointestinal tract, and cardiovascular system