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These functional results suggested that the analgesic effects of SHU 9119 are e/e Mc1r independent and likely due to ligand effects on MC4R or MC3R, consistent with the possibility that MC4R or MC3R signaling may balance opioid signaling to modulate nociceptive thresholds

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Nayan Patel: And that's an old, this was actually done at the NIH back in the late 80s, early 90s

Variants in the GLP1R rs6923761 site, for example, influence receptor sensitivity and may affect optimal dosing strategy

the former can significantly impair patients quality of life, whereas the latter can threaten life itself