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In the presence of elevated levels of glucose and fatty acids, insulin gene transcription inhibition is due, at least in part, to decreased expression and function of the transcription factors MafA and pancreatic duodenal homeobox-1 (PDX1) [9]

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Finally, although most of the evidence of a potential benefit of GLP-1 RAs in HF originates from CVOTs in type 2 diabetes, it may likely be in the weight management setting that the drug class could be of the highest clinical value, particularly in people with HFpEF