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16 , 17 The M1 phenotype is associated with the release of cytokines and mediators that amplify inflammation and promote neurotoxicity, whereas the M2 phenotype suppresses inflammation and fosters tissue repair

doi:10.4161/spmg.26391

Furthermore, studies undertaken in vitro using isolated mouse hepatocytes have revealed that AMAP has a markedly reduced propensity to cause cytotoxicity when compared with acetaminophen, even though AMAP treatment results in higher levels of covalent binding to proteins (Holme et al