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Senescent cells are known for their pro-inflammatory secretory phenotype, termed the Senescence-Associated Secretory Phenotype (SASP), which FOXO4-DRI effectively reduces, improving tissue function

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The peptide activates extracellular signal-regulated kinase 1/2 (ERK1/2) phosphorylation in a dose-dependent manner, leading to nuclear translocation and activation of transcription factors including c-Fos, c-Jun, and early growth response-1 (Egr-1) that drive expression of genes involved in cell growth, migration, and angiogenesis

Available at: Summary Keywords Parkinsons disease, Lewy body dementia, Alzheimers disease, melanin, glyphosate, inflammation, amyloid plaque, gut microbiome Citation Berg SZ and Berg J (2023) Melanin: a unifying theory of disease as exemplified by Parkinsons, Alzheimers, and Lewy body dementia