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Moreover, elderly pDCs are less capable of stimulating CD8 + T cells to produce perforin and granzyme, which are crucial for cytotoxic activity, and they are less efficient in promoting IFN- secretion by CD4 + and CD8 + T cells [258]

Moreover, the phenotypic consequences of these variants may only emerge under conditions of physiological stress, such as rapid growth, inflammation, or pharmacologic challenge

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However, investigation of the effect of UA on such models is limited by the profound differences between humans and e.g., rodents in UA metabolism, primarily due to the loss in humans of a functional uricase