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Conversely, excessive levels of oxidants in skeletal muscle, often linked to prolonged exercise, can impair proteins critical to excitationcontraction coupling, thereby inducing muscle fatigue and diminishing the physiological advantages of ROS production during moderate-intensity exercise (58)

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MOTS-c interacts synergistically with exercise intervention to regulate PGC-1alpha expression, attenuate insulin resistance and enhance glucose metabolism in mice via AMPK signaling pathway

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