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Yes many of our founding-100 reservations come from patients who started on a scale-first platform and want a more clinical relationship

Preclinical research links it to three main mechanisms: VEGFR2/eNOS angiogenic signaling, Src-caveolin-1-eNOS nitric oxide modulation, and FAK/paxillin cell-migration signaling

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moreover, patients with high expression of CALD1, CRABP2, DHRS7 and RIMS1 revealed worse prognostic outcomes (Fig