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The two peptides have complementary mechanisms of action, and preclinical studies suggest potentially additive healing effects across musculoskeletal and connective tissue models

Improved stability does not prove better disease, symptom or longevity outcomes

The disruptive nature of enterotoxins on TJ complexes can also lead to the movement of basolateral proteins to apical sites on epithelial cells where they may be utilised by pathogens or their toxins as alternative receptors facilitating infection (Muza-Moons et al

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