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miR-19a contributes to gefitinib resistance and epithelial mesenchymal transition in non-small cell lung cancer cells by targeting c-Met

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We now demonstrate that GIPR agonist administration critically relies on Gipr expression in the hindbrain, while GIPR antagonism relies on hypothalamic Gipr expressionboth are probably neuronal populations, given that we used hSyn-driven Cre-expression for the region-specific knockout studies

Moreover, the lower plasma cobalamin levels also were associated with the presence of symptoms consistent with major depressive disorder

Microbial ecology: human gut microbes associated with obesity (in Eng)