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Here, we showed that BHBA enhanced brain-derived neurotrophic factor (BDNF) expression by increasing H3K4me3 and decreasing H2AK119ub occupancy at the Bdnf promoters I, II, IV, and VI in hippocampal neurons

Researches about gut microbial of the influence of IF on subjects who have MS were expected as a result of migration of intestine immune cell subsets

As discussed above (in the section Role of mitochondria in synaptic transmission), Ca 2+ signaling is involved in the accumulation of mitochondria at the postsynaptic regions, where these organelles participate in neuronal Ca 2+ buffering and support of neurotransmission [93]

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SSG as an inflammasome checkpoint SSG, as a thiol-based switch, links GSH metabolism to inflammasome licensing