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In two studied groups of healthy aged woman, LC supplementation alone [16, 17], or in combination with L-leucine [18], induced an increase of fasting plasma TMAO levels

In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis

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Oral MNMs with multiple functions may represent a promising future development trend ( Fig