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Objectives: This review synthesizes current evidence on EED impacts on male reproductive health, focusing on molecular mechanisms, population-based evidence, transgenerational effects, and intervention strategies

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Through glucose-mimicking surface glycosylation, the nanocarrier achieved selective uptake by tumor cells via GLUT1 and bone marrow-derived suppressor cells (MDSCs) via GLUT3, respectively, enabling concurrent chemotherapy and metabolic suppression of immunosuppressive myeloid cells

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The specific mechanism of the Nrf2/HO-1 signaling pathway in regulating ferroptosis is still unclear and requires further investigation