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Consistent with Oga deletion, pharmacological inhibition of Oga, which induces acute elevation of O-GlcNAcylation, has been shown to attenuate hippocampal activity by diminishing the intrinsic excitability of CA1 neurons and excitatory synaptic transmission at Schaffer collateral-CA1 synapses [80]

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[6] Research [edit] Pre-clinical research has indicated that BPC-157 may have cytoprotective, neuroprotective, and anti-inflammatory effects, and may also accelerate tissue and organ healing

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SAG S-acetyl-glutathione SAG S-acetyl-glutathione a delayed release delivery system designed to protect the molecule from degradation by gastric acids in the stomach and carry the SAG to the small intestine where it can be readily taken up intact by chylomicrons in the gut