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The need for neuroprotection in AD is underscored by the diseases long preclinical phase, during which silent pathological processes accumulate before any measurable symptoms emerge

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This may result from severe disruption of functional capacity in the gut microbiota of SZ patients, leading to a marked increase in pro-inflammatory metabolites and a significant decrease in anti-inflammatory metabolites (71)

Correlation analysis revealed that SLC7A11, HMOX1, G6PD, RRM2, KIF20A, HELLS, SPP1, CCR2 and DCN were positively correlated with plasma cells and CD8+ T cells, whereas GPT2, GLS2, IRF1, and IGF1 were negatively correlated with these cell subsets (Fig