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In the present study, l-carnitine supplementation induced glucose sparing by normalising FA utilisation, while d-carnitine-fed tilapia used more glucose and protein in response to a lack of energy derived from lipids

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It is 50% identical to GLP-1 with a nine-residue C-terminal extension, and is of more potential pharmacological interest due to its higher binding affinity to GLP-1R [9]