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Acute myeloid leukemia cells educate mesenchymal stromal cells toward an adipogenic differentiation propensity with leukemia promotion capabilities

Impaired ribosomal biogenesis by noncanonical degradation of beta-catenin during hyperammonemia

Our model also accounts for the efficacy of commonly-used treatments of CVS, including mitochondrial-targeted therapies, propranolol (beta blockade interferes with catecholamine-induced release of calcium via the RYR2 calcium channel (101), and amitriptyline (used for peripheral neuropathy)

Sabbatini M, Bona E, Novello G, Migliario M, Reno F