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FNMTC, Familial non-medullary thyroid carcinoma

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Importantly, the formation of this novel metabolite could help explain previously observed effects of THI on certain cellular systems, including the blocking of calcium channels which play an important role in the homeostasis of Ca 2+

10.1186/s12933-020-01071-y 126 OuY

Liu and colleagues analyzed a cell model of AMD induced by sodium iodate (SI) and reported that SI can consume intracellular GSH, liberate Fe 2+ from liposomes, increase cellular ROS levels, and increase lipid accumulation, leading to ferroptosis in ARPE-19 cells (244)