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This premature polyploidy in Ercc1 -deficient liver is most likely caused by increased levels of p21 in response to accumulating DNA damage (Chipchase, ONeill, and Melton 2003)
The peptide GH secretagogues fall into two functional groups: the ghrelin-mimetic GHRPs (which, like MK-677, act at GHSR1a) and the GHRH analogs (which act at the separate GHRH receptor)
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Furthermore, the upregulation of transforming growth factor (TGF)- and WNT signaling pathways promotes the activation of SNAIL and DLX2, which in turn upregulate the expression of GLS [112]
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