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Autoimmune demyelinating disease models (e.g., MS): By dampening inflammatory processes involved in myelin damage, TB500 Fragment (1723) has been proposed as a tool to study prevention of demyelination and relapse dynamics in multiple sclerosislike models

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Thus, mitochondrial translation of MOTS-c would lead to tandem start and stop codons [4]

This unusual resilience extends to other harsh environments as well, which may contribute to its effectiveness when applied to wound sites where pH levels and enzyme activity can be unpredictable