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doi: 10.1158/0008-5472.CAN-15-1551

(2021) established a mouse model and observed that the inhibition of TMAO production alleviated CKD in mice

We observed a positive correlation between cyclin E1 and TMLHE protein expression in two different tissue microarrays (TMAs) of serous ovarian cancers (Fig

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Glycine and N-acetylcysteine (GlyNAC), a GSH precursor complex, can exogenously replenish glycine and cysteine levels and was reported to attenuate oxidative stress-induced damage and delay aging [16]