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2024;31(4):266274 Kennedy C, Hayes P, Salama S, Hennessy M, Fogacci F

We propose the hypothesis that the current body of evidence is consistent with a prevention-focused model, wherein semaglutides primary value may lie in modifying the upstream metabolic and inflammatory drivers of neurodegeneration, such as those prevalent in vascular and metabolic cognitive impairment, rather than reversing established amyloid-driven AD pathology

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