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The disproportionate energy needs and reliance on glucose makes the brain particularly vulnerable to disruptions in the regulation of energy metabolism [192, 193]

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This article reviews the problem of AIWG and AP metabolic side effects in pediatric populations, proposed mechanisms underlying this serious side effect, and strategies to mitigate adverse impact

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Neuroprotective and cytoprotective compounds Neuroprotective and cytoprotective compounds have gained prominence as potential therapies to reduce secondary brain injury, which is largely mediated by excitotoxicity, oxidative stress, apoptosis, and BBB disruption