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CRISPR-Cas9 knockout screen confirms the role of LCS3 in mediating antioxidant response and reveals suppression of NQO1 as a mechanism of resistance To complement the effort to identify targets of LCS3 with TPP and to interrogate potential mediators of the LCS3 response systematically, we performed a genome-wide, loss-of-function CRISPR-Cas9 screen in LCS3-sensitive LUAD cells

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Suppressed T helper 2 immunity and prolonged survival of a nematode parasite in protein-malnourished mice

Angiotensin type 1 receptor antagonist inhibits lipopolysaccharide-induced stimulation of rat microglial cells by suppressing nuclear factor B and activator protein-1 activation: Lipopolysaccharide-induced stimulation of rat microglial cells

It stimulates the activity of enzymes and growth factors involved in tissue remodeling and regeneration, helping to maintain youthful skin and promote faster recovery from injury