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This mechanism reveals the intricate, multi-layered interactions between metabolic dysfunction, immune activation, and neuroinflammatory damage, providing a potential molecular basis for the subsequent development of neuroinflammation ( Figure 3 )

ROS generation was analyzed by MitoSox and endoplasmic reticulum stress markers were determined using qPCR and Western blot

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The phospholipid structure of liposomes allows them to integrate with cell membranes through a process called membrane fusion

The impaired wound healing response observed in smokers can also hinder hepatic regeneration, thereby exacerbating fibrosis and increasing the likelihood of replication errors and de novo mutations, potentially linking to carcinogenesis [47, 48]