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The inflammation associated with acute pancreatitis is thought to damage nerve pathways controlling digestive function, particularly affecting the stomach muscles responsible for moving food to the small intestine

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This could explain the various and efficient antioxidant systems at muscle level including various isoenzymes of Superoxide dismutase (SOD), GPx and catalase, high activity of GSH-dependent enzymes and a strong ability to synthesize new GSH [24,50]

Statistically significant prevention of the decrease in SOD and CAT activities due to the use of 120 mg/kg HCQ in the ATP treatment group was found when compared to the HCQ only group