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The most common NRAS mutation is an amino acid substitution at position 61 (Q61K, Q61L and Q61R) [16]

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IELs protect the epithelial barrier by promoting pathogen clearance and lysing pathogen-infected cells (Yusta et al., 2015)

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In 2021, a research team led by Professor Peter Arthur Calabresi, Director of the Multiple Sclerosis Center at Johns Hopkins University School of Medicine, published findings in the international journal Neurotherapeutics (Impact Factor: 6.9) indicating that NLY01 significantly slowed the progression of MS in animal models by reducing neuroinflammation and protecting nerve cells