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Next, we evaluated the excretion, metabolism, and tissue distribution of BPC157 in rats after a single IM injection of 100 g/300 Ci/kg [ 3 H]BPC157

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Gene knockdown experiments further revealed that SiO-induced ferroptosis and ROS elevation are mediated by NOX4 upregulation and Smad3 activation, highlighting the critical role of ferroptosis and this signaling axis in SiO-induced intraocular pressure elevation

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