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Our framework will integrate genomic variants summarized as polygenic risk scores (PRS), bulk or single-cell transcriptomes, targeted proteomic panels, metabolomic/lipidomic signatures, and clinical/phenotypic features from electronic health record (EHR)/real-world data (RWD)

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doi:10.1016/j.ajpc.2020.100005 Association OM

Mechanisms linking environmental agents to the development of Autoimmune Diseases Oxidative stress-mediated disturbance of Nrf2, induction of NF-kB, PARP1, and NLRP3 can cause activation of both innate and adaptive immune systems, resulting in pro-inflammatory cytokines and production of autoantibodies, leading to tissue damage in Autoimmune disease