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Ultimately, by inhibiting abnormal proliferation of PASMCs and endothelial cells, reducing extracellular matrix deposition, and improving myocardial energy metabolism patterns, it achieves the therapeutic goal of delaying pulmonary vascular remodeling and the progression of right heart failure, providing a unique multi-dimensional regulatory strategy of natural drugs for PH treatment

Our results indicated that all the fourteen selected acyl-carnitines with FDR q 1.0 (i.e., C2, C8, C10, C10:1, C10:2, C12, C12:1, C14:1, C14:1-OH, C14:2, C14:2-OH, C16:2, C16:2-OH, and C18) were lower in posttreatment patients after controlling age, sex, BMI, smoking, and drinking (Tables 3 and 4)

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This flexibility is not merely a convenience