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The GBA effects its influence on depression through neuroinflammatory pathways alteration of neurotransmitter precursors and the secondary dysregulation of HPA axis

Perturbations of metabolic status within the pathway as ROS-processing activity increases could contribute to oxidant-triggered signalling

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Simultaneously, the medication slows gastric emptying

The resulting elevation in intracellular Ca activates protein kinase C (PKC), which subsequently phosphorylates and activates MEK, ultimately leading to enhanced phosphorylation and activation of ERK1/2 within the MAPK pathway [51] [52](Fig