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It recognizes N-terminal truncated A modified with pyroglutamate at position 3 (N3), a form of A that is aggregated into amyloid plaques

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However, when antioxidative systems become saturated, RBCs accumulate oxidative lesions, provoking biochemical alterations and advancing pathological developments ( 2 O 2 can diffuse out of erythrocytes and provoke inflammatory activation in the lungs microvascular endothelium (Kiefmann et al., 2008)