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Paradoxically, activating the glucagon receptor in a controlled, pulsatile pharmacological fashion appears to increase energy expenditure, enhance fat burning in the liver, and promote thermogenesis, effects that purely GLP-1-based agents do not reliably produce

Personalized dietary advice could help patients set realistic expectations about their outcomes, as GLP-1 medications are not cure-alls, and significant lifestyle changes may be beneficial

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Cells that are defective in KEAP1 have increased NRF2 transcription and increased import of cystine and export of glutamate, thus requiring more import of glutamine from the milieu to regenerate intracellular glutamate via glutaminase activity

Hence, ME/CFS significantly reduces a patients quality of life (11, 24, 2931), impacting activities of daily living, socialising, family life, emotional wellbeing, work, and education (11, 23)