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The combination of selenocysteine and 5-fluorouracil can regulate the intracellular redox system, induce cellular oxidative stress and DNA damage, and block the extracellular signal-regulated kinase (ERK) signaling pathway, thus promoting 5-fluorouracil-induced apoptosis of tumor cells, and enhancing its antitumor effects (147)

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Because GLP-1 medications naturally reduce appetite and slow digestion, people often eat less overall

As a major form of glomerulonephritis, LN represents one of the most severe organ manifestations of systemic lupus erythematosus (SLE) [37]