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While dihexa at 1 nM and 10 pM alone does not activate c-Met, it markedly augments the capacity of HGF at 1.25 and 2.5 ng/mL to activate c-Met [1]

however, a subsequent double-blinded, placebo-controlled trial failed to replicate these benefits and even noted exacerbation in some instances

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Proper patient selection-including avoidance in those with active peptic ulcer disease, advanced renal impairment, uncontrolled bleeding disorders, or hypersensitivity to aspirin-like drugs-optimizes the benefit-to-risk profile