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[DOI] [PubMed] [Google Scholar] 147.Vivarini .D.C., Silva T.C.C., Saliba A.M., Boaventura V.S., Frana-Costa J., Khouri R., Dierckx T., Teixeira K.L.D., Fasel N., Barral A.M.P., et al

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Proposed mechanisms include upregulation of growth-hormone-receptor expression in injured tissue, VEGF-driven angiogenesis, modulation of the nitric-oxide pathway in vascular endothelium, and activation of FAK-paxillin signaling